not doppler home sheep home 2 lost in london

Obstetric technologies and therapies are also directed toward general pathophysiologic processes (such as uterine and uteroplacental Doppler velocimetry, anticoagulant therapy, and maternal immunization and intravenous gamma globulin) rather than specific lesions or tissue diagnoses. It is illogical to expect that the placenta and its diseases should be any more or less complicated than diseases of the liver and kidney. This comprehensive approach yields not only a summary “label” for the histopathologic findings but also information regarding etiology, prognosis, and optimal therapy. Finally, the pattern is correlated with a variety of clinical data, laboratory data, and additional pathologic studies to produce the final clinical/pathologic diagnosis. Neither hepatocellular necrosis nor glomerulosclerosis, for example, is diagnostic of one and only one hepatic or renal disease in those diseases, individual lesions are considered as part of a greater histopathologic pattern. This would be an unrealistic goal, because in no other organs are such relationships the rule. One important limitation is the inability to make a one-to-one link between any one placental or uteroplacental vascular lesion and a particular maternal or fetal/neonatal problem. For that reason, we have directed our efforts toward clarifying the contributions and limitations of placental examination to clinical practice. Placental pathologic examinations are pointless if they do not provide clinically useful data. Obstetric endorsement of the utility of placental histologic examination is commonly lukewarm, especially from obstetricians who do not have a placental pathologist as part of their own local clinical care team. The role of the placental pathologist in clinical obstetrics and neonatology has long been controversial.

Not doppler home sheep home 2 lost in london